Cameron Kyle-Sidell, MD

@cameronks

Critical Care Medicine, Emergency Medicine

Brooklyn, NY
EMHUM.com
Joined April 2011
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  1. Retweeted
    Mar 8

    🫁COVID-19 Pathophysiology: Are Platelets and Serotonin hiding in plain sight? 📜Pre-print 🔗 📍Rationale laid out for why 5HT-2 antagonism helps reverse lung + systemic injury in Asking for consideration in 💊ACTIV 🩸RECOVERY 🏥WHO 🧵Retweet🧵

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    9 replies 49 retweets 103 likes
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  2. Retweeted
    Mar 2

    We practice resource unlimited medicine in the US, and in March and April of 2020 in NYC, we ran into limited resources and didn’t change our practice. That is part of the COVID story of this city in its darkest hours. 5/5

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  3. Mar 2

    We practice resource unlimited medicine in the US, and in March and April of 2020 in NYC, we ran into limited resources and didn’t change our practice. That is part of the COVID story of this city in its darkest hours. 5/5

    2 replies 8 retweets 22 likes
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  4. Mar 2

    Disparities exist between times, circumstances, and places. When for ex. a highly resourced Harvard hospital recommends early intubation everywhere when a peak is happening somewhere else, they are not recognizing this. 4/5

    1 reply 6 retweets 13 likes
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  5. Mar 2

    I have worked at hospitals that are exceedingly adept at protecting against vent induced injury, and some that are less so. Typically has to do with how many skilled persons are involved in a patients’s care. (As opposed to the skill of a person) 3/5

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  6. Mar 2

    Every school does not provide the same level of education, and it is likely not the teachers fault. Some teachers have 30 students, some have 10 with 2 teacher’s assistants. 2/5

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  7. Mar 2

    The notion that there is a one-size all answer to the “timing of intubation” needs to be extinguished. In the PSILI vs VILI decision tree, we need to accept that the ability to protect against vent injury is not the same in every time period, hospital, city, or country. 1/5

    Late intubation associated with longer ICU LOS and duration of MV in . Nonsurvivors had longer time to intubation vs survivors. Read more in the latest issue of :
    6 replies 11 retweets 29 likes
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  8. Mar 1

    COVID testing is easily available in NYC now, but I vividly remember the end of Feb and early March when a busy doc had to make a long phone call to (hopefully) get a patient tested—result not for days—for a disease we’d never seen. A nice article about that confusing time 👇👇

    One year from the front lines of COVID-19 in NYC, these are my thoughts on our local pandemic preparedness and response, what went wrong at the operational level, and how to make it better. Thank you , :
    2 replies 8 retweets 29 likes
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  9. Retweeted
    Feb 27

    ☑️𝙨𝙚𝙫𝙚𝙧𝙚 𝙋𝙡𝙖𝙩𝙚𝙡𝙚𝙩 𝘼𝙘𝙩𝙞𝙫𝙖𝙩𝙞𝙤𝙣 is a hallmark feature of COVID19: 👉𝗣𝗚𝗜𝟮 𝗲𝗹𝗲𝘃𝗮𝘁𝗶𝗼𝗻 is pulm vasculature's dire attempt to counteract the diffuse vasoconstriction caused by PLT 𝘀𝗲𝗿𝗼𝘁𝗼𝗻𝗶𝗻

    1 reply 21 retweets 40 likes
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  10. Retweeted
    Feb 26

    Now published in ERJ! Pulmonary Vascular Abnormalities as measured by FRI independently and accurately predict mortality and the need for intubation in :

    1 reply 13 retweets 51 likes
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  11. Retweeted
    Feb 24

    Climate? ‘s got you. Please RT and lets help him spread the good word on science!

    7 retweets 8 likes
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  12. Retweeted
    Jan 29

    This is an important study by and colleagues on platelets & serotonin in COVID19.

    1 reply 42 retweets 88 likes
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  13. Retweeted
    Jan 29

    Platelet hyperreactivity (to thrombin) is significantly 𝗵𝗶𝗴𝗵𝗲𝗿 in COVID ARDS vs. non-COVID ARDS. And naturally: Plasma 𝘀𝗲𝗿𝗼𝘁𝗼𝗻𝗶𝗻 in COVID ARDS >> non-COVID ARDS. Outstanding work by et al.

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    9 replies 66 retweets 129 likes
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  14. Retweeted
    Jan 25

    I spent November and December talking to nurses across the country, documenting new ways they are taking care of patients—and each other. Even amid record numbers of hospitalizations, they are reshaping COVID care 1/ For :

    1 reply 13 retweets 23 likes
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  15. Jan 23

    Time to change the paradigm in which we frame this terrible disease. An “immunothrombotic” rather than “pneumonia” paradigm will help frame the right questions moving forward. Such as: are platelet mediators driving the process?

    Have reviewed a lot of papers on clots in severe COVID-19 in last weeks & most ignored immunothrombosis as cause of segmental/subsegmental changes on CTPA & counted them as “PE” It’s time to for all to acknowledge this phenomenon & move on!
    5 replies 44 retweets 104 likes
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  16. Retweeted
    Jan 22
    Replying to and

    Agree with that platelet mediators are key in secondary effects. Have to consider treating similar to ACS. Several centres trying antiplatelets in addition to anticoagulation.

    1 reply 7 retweets 7 likes
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  17. Retweeted
    Jan 22

    This slide should be read so many times until the entire slide makes perfect sense. That’s the only way forward to understand the severe lung injury of COVID19. Thank you for this early contribution. This and work are outstanding!

    , and
    2 replies 27 retweets 66 likes
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  18. Retweeted
    Jan 22

    If I were a betting man, I would not bet against below (or me) in this pandemic: - Anticoagulation saves lives - Antiplatelets will save lives - Severe COVID is a FULL_BLOWN SEROTONIN TOXICITY - Cyproheptadine is a MUST in severe COVID - Fentanyl is HARMFUL

    Important announcement from ATTACC, ⁦⁦⁩ and ⁦⁩: Full-dose blood thinners decreased need for life support and improved outcomes in hospitalized COVID-19 patients
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    6 replies 49 retweets 92 likes
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  19. Jan 15

    The author (Tobin) writes: “Evidence-based guidelines can never help a doctor resolve whether a patient is best managed by inserting an endotracheal tube or avoiding its use. That decision is based on clinical judgment, gestalt, and tacit knowledge.”

    related , debate goes on! Typical or atypical?? question about atypicality matters to researchers, for clinicians is distracting: critical bedside dilemma ➡️ whether to intubate or not! Referred to et al
    3 replies 18 retweets 66 likes
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  20. Jan 15

    Here is on the outsized role of SEROTONIN in severe COVID19 disease. A truly, exceptionally IMPORTANT thread 👇

    🧵on serotonin, SSRIs, serotonin-blockers, and COVID19 All causes of ARDS are associated with activation of resident lung platelets and megakaryocytes. The degree to which this activation occurs differs between various etiologies of ARDS, as demonstrated in ...
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